
Postdoc
Department of Dermatology
Publications
Commensal myeloid crosstalk in neonatal skin regulates long-term cutaneous type 17 inflammation.
bioRxiv : the preprint server for biology
FLG Deficiency in Mice Alters the Early-Life CD4+ T-Cell Response to Skin Commensal Bacteria.
The Journal of investigative dermatology
Intestinal inflammation alters the antigen-specific immune response to a skin commensal.
Cell reports
Baby's skin bacteria: first impressions are long-lasting.
Trends in immunology
Regulatory T cells promote innate inflammation after skin barrier breach via TGF-ß activation.
Science immunology
Modification of the pulmonary MyD88 inflammatory response underlies the role of the Yersinia pestis pigmentation locus in primary pneumonic plague.
Infection and immunity
Multi-Immune Agonist Nanoparticle Therapy Stimulates Type I Interferons to Activate Antigen-Presenting Cells and Induce Antigen-Specific Antitumor Immunity.
Molecular pharmaceutics
Developing Human Skin Contains Lymphocytes Demonstrating a Memory Signature.
Cell reports. Medicine
Toxin-Triggered Interleukin-1 Receptor Signaling Enables Early-Life Discrimination of Pathogenic versus Commensal Skin Bacteria.
Cell host & microbe
Yersinia pestis exploit early activation of myeloid differentiation primary response 88 (MyD88) for growth in the lungs during pneumonic plague.
Infection and immunity
Skin Commensal Antigens: Taking the Road Less Traveled.
Trends in immunology
Induction of Type I Interferon through a Noncanonical Toll-Like Receptor 7 Pathway during Yersinia pestis Infection.
Infection and immunity
Bacterial programming of host responses: coordination between type I interferon and cell death.
Frontiers in microbiology
Bacterial Activation of Type I Interferons/Yersinia Activation of Type I Interferon
Bacterial Activation of Type I Interferons/Yersinia Activation of Type I Interferon
Early apoptosis of macrophages modulated by injection of Yersinia pestis YopK promotes progression of primary pneumonic plague.
PLoS pathogens
Host stress and immune responses during aerosol challenge of Brown Norway rats with Yersinia pestis.
Frontiers in cellular and infection microbiology